Cardiovascular

Cardiac Sarcoidosis: Key Concepts in Pathogenesis, Disease - download pdf or read online

By Andrew M. Freeman, Howard D. Weinberger

ISBN-10: 3319146238

ISBN-13: 9783319146232

ISBN-10: 3319146246

ISBN-13: 9783319146249

This ebook will offer, for the 1st time on hand, a concise yet excessive yield subject overview of cardiac sarcoidosis from threat components to the improvement of the ailment via cures. The ebook will comprise techniques which are rising and those who at the moment are identified at the subject, and may use actual international examples to aid illustrate top practices within the administration of this affliction entity.

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Extra info for Cardiac Sarcoidosis: Key Concepts in Pathogenesis, Disease Management, and Interesting Cases

Example text

The cardiac FDG images show good suppression of FDG from the myocardium with no focal uptake involving the left or right ventricles 44 R. Blankstein and S. Dorbala Most patients with cardiac involvement will exhibit a focal or focal-on-diffuse area of myocardial FDG uptake. Less frequently, focal areas of increased FDG uptake involving the right ventricle may also be seen, a finding which is associated with adverse prognosis [9]. In earlier stages of disease isolated FDG uptake may be seen without any perfusion defects.

Features of pulmonary sarcoidosis include mediastinal and hilar lymphadenopathy, nodularity, centrilobular nodularity, reticular abnormality, ground glass attenuation, perihilar confluent opacities, architectural distortion and pulmonary fibrosis. [18F]fluorodeoxyglucose (FDG) positron emission tomography (PET) whole body imaging can be used for staging, identification of occult sites of disease and identification of sites suitable for biopsy [11]. Limitations of Cardiac MRI in the Diagnosis of Cardiac Sarcoidosis Cardiac MRI is not a specific imaging modality for cardiac sarcoidosis and attention to correct MRI protocols and technique as well as artifacts and differential considerations is important.

Uptake of FDG by the lateral wall may represent a normal variant, particularly when (a) there is no perfusion defect involving the lateral wall; (b) the FDG uptake is homogenous (as opposed to patchy); and (c) the likelihood of cardiac disease is low. 6. Patients with advanced cardiac sarcoidosis may have “burnt out” disease with no (or little) FDG uptake. Such patients are more likely to have left ventricular systolic dysfunction. Patients with large amount of scar and reduced ejection fraction are less likely to have an improvement in ejection fraction following immunosuppressive therapy.

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Cardiac Sarcoidosis: Key Concepts in Pathogenesis, Disease Management, and Interesting Cases by Andrew M. Freeman, Howard D. Weinberger


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