Download e-book for kindle: Comprehensive Gynecology (6th Edition) by Gretchen M. Lentz, Roger A. Lobo, David M. Gershenson, Vern

By Gretchen M. Lentz, Roger A. Lobo, David M. Gershenson, Vern L. Katz

Entire Gynecology provide you with whole, well timed, and simple entry to important info on the entire scientific and surgical matters affecting your perform. no matter if you're a resident or practioner, you'll get all of the functional, in-depth assurance you must remain on the leading edge of your box. Now in its 6th version, this center reference, previously edited via Drs. Droegemueller, Stenchever, Mishell and Herbst, remains to be your fundamental source for in-depth and updated information.

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Comprehensive Gynecology is the must-read textual content for either citizens and tested practitioners.

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Extra info for Comprehensive Gynecology (6th Edition)

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Depending on the ethnic group of the mother and father, the risk for a child having Figure 2-14 Pedigree illustrating autosomal recessive inheritance. Here the parents of the affected children are first cousins, as denoted by the double line connecting them. ) 2 Table 2-2 Carrier Frequencies for Cystic Fibrosis in Different Populations Ethnicity Chance of Being Carrier Chance Both Carriers* European descent Hispanic American African American Asian American 1 1 1 1 1 in 1 in 1 in 1 in in 29 in 46 in 65 in 90 841 2,116 4,225 8,100 *The chance for an affected child being born to these couples is the chance that both are carriers times ¼.

The viability of the genetically unbalanced gametes depends on the chromosomes involved in the reciprocal translocation, the size of the translocated chromosome material, and the sex of the carrier. In addition, most reciprocal translocations are unique to a family, and, consequently, the reproductive fitness of the carrier depends on the 2 Reproductive Genetics Table 2-5 Karyotypes discovered in Subjects with Phenotypic Characteristics of Turner Syndrome Figure 2-18 A, Photo of a 20-week fetus with Turner syndrome, 45, X.

The condition is due to an expansion of the triplet repeat CGG located in the untranslated region of the first exon of the gene called FMR1 (fragile X mental retardation 1). The triplet expansion blocks normal function of the FMR1 gene, thus causing the syndrome. Normal individuals have about 8 to 50 copies of the CGG triplet, whereas affected individuals have from 200 to more than 1000 copies. Individuals with an intermediate number of copies (52–200) are known as premutation carriers; this level of “expansion” renders the triplet-repeat segment unstable.

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Comprehensive Gynecology (6th Edition) by Gretchen M. Lentz, Roger A. Lobo, David M. Gershenson, Vern L. Katz

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